Dr. Swarupa Panda

Assistant Professor

Dr. Swarupa Panda completed her master’s in biotechnology from Utkal University, India, where she developed a keen interest in cellular molecular mechanisms. Following her M.Sc., she worked as a Junior 亚洲通_亚洲通官网 Fellow at the Center for Cellular and Molecular Biology (CCMB) in Hyderabad, gaining essential experience in immune regulation and host-pathogen interactions, which laid the foundation for her Ph.D. at Ume? University, Sweden. Under Prof. Nelson Ongondo Gekara’s mentorship, she discovered MYSM1 as a key regulator of inflammation, offering new therapeutic targets for inflammatory diseases. As an EMBO Long-Term Fellow at Oslo University Hospital, Norway, Dr. Panda further advanced her research, exploring autophagy, metabolism, and ferroptosis in cancer. Her work identified SLC7A9 as a critical transporter that mitigates ferroptosis in oxidative stressed tumors, presenting new cancer treatment strategies. Throughout her career, Dr. Panda has also been actively involved in teaching courses like Immunology, Transgene Technology, and Advanced Cancer Biology, mentoring the next generation of scientists. As an assistant professor, Dr. Panda continues to focus on ferroptosis, peroxisomal dysfunction, and immune regulation in health and disease. Her work, published in high-impact journals like Nature Communications and Immunity, has significantly contributed to our understanding of immune modulation, tumor growth, and cellular stress responses. She remains dedicated to developing novel therapeutic strategies and advancing cutting-edge research in her field.

Department of Ageing 亚洲通_亚洲通官网

CURRENT ACADEMIC ROLE & RESPONSIBILITIES

    Dr. Swarupa Panda?is Assistant Professor, Department of Ageing 亚洲通_亚洲通官网 at 亚洲通_亚洲通官网 School of Life Sciences.

AREAS OF INTEREST, EXPERTISE AND RESEARCH

Area of Interest

Host-Pathogen Interactions, Ferroptosis Regulation in Health and Disease, Redox Homeostasis Disruption, Ubiquitin System ,Cancer Cell Metabolism, Inflammation-Induced Cell Death, Therapeutic Targets in Ferroptosis